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Figure 2: (a) Sagittal T1WI(TE14/TR406), (b) sagittal T2WI(TE115/TR4000), (c) axial T1WI(TE14/TR478), (d) axial T2WI(TE147/TR4780), (e) sagittal T1WI with fat suppression and intravenous contrast(TE10/TR511), (f) axial T1WI with fat suppression and intravenous contrast (TE11/TR562), and (g) sagittal gradient echo (TE18/TR600). They showed an intramedullary space occupying lesion that involves D11 and D12 segments consecutively and showed low-signal intensity in T1WI and increased heterogeneous signal intensity in T2WI with associated syrinx that reaches the D1 level. Post-contrast administration showed a faint enhancement pattern with associated hemorrhagic components that showed a blooming artifact indicating hematomyelia

Figure 2: (a) Sagittal T1WI(TE14/TR406), (b) sagittal T2WI(TE115/TR4000), (c) axial T1WI(TE14/TR478), (d) axial T2WI(TE147/TR4780), (e) sagittal T1WI with fat suppression and intravenous contrast(TE10/TR511), (f) axial T1WI with fat suppression and intravenous contrast (TE11/TR562), and (g) sagittal gradient echo (TE18/TR600). They showed an intramedullary space occupying lesion that involves D11 and D12 segments consecutively and showed low-signal intensity in T1WI and increased heterogeneous signal intensity in T2WI with associated syrinx that reaches the D1 level. Post-contrast administration showed a faint enhancement pattern with associated hemorrhagic components that showed a blooming artifact indicating hematomyelia